By Antony F. McDonagh (auth.), Carl A. Goresky M.D., Murray M. Fisher M.D. (eds.)
ISBN-10: 1468426494
ISBN-13: 9781468426496
ISBN-10: 1468426516
ISBN-13: 9781468426519
Jaundice is far greater than a scientific signal of liver illness. it's also a pathophysiological sickness. via learning it now we have come to a higher figuring out of ways the liver features below general and irregular conditions. despite numerous vital advances during this box, it has now not lately been the topic of a finished and interdisciplinary evaluate. This Symposium was once held in Montreal on may well 31 and June 1, 1974, and the specialists who participated in it got here jointly for the aim of reviewing the present prestige of Jaundice. The Editors truly get pleasure from the phenomenal contribution which those specialists made to the second one foreign Symposium of the Canadian Hepatic origin. also they are really indebted to Valerie M. fee, govt Director, Canadian Hepatic starting place, for her helpful function within the instruction of this ebook. Carl A. Goresky Murray M. Fisher v Contents BILIRUBIN CHEMISTRY an summary of Bilirubin Chemistry 1 A. F. McDonagh The Conjugates of Bilirubin 19 E. R. Gordon dialogue interval. • • 39 R. Schmid, Char:imm BILIRUBIN PRODUCITON forty three Bilirubin Metabolism: an outline • • • R. Schmid Bilirubin creation fram Non-Erythroid SoUI"Ces • • • • • • • • • • • • • • • • fifty seven S. H. Robinson Bilirubin creation fram Erythroid resources sixty nine U. Muller-Eberhard and E. F. Johnson Induction Mechanisms for Bile Pigment Formation eighty five B. A.
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1). As the labeled cells left the circulation and were destroyed, a large late peak of labeled bile pigment production was observed. Measurements made during the first few days also revealed an "early-labeled peak" of pigment production which preceded the entry of significant numbers of labeled red cells into the peripheral blood. Under normal 57 58 S. H. -... -" ...... 3 ..... Z \ NORMAL MAN ··\. 1 ~ ~ '. \ \ .... W a::: o ..... ; , '~\---HEMIN u Q. :' O~~ o __~__L - - L_ _~~_ _~_ _L - - L_ _~~_ _~ 20 40 60 80 100 120 140 160 180 200 220 240 TIME IN DAYS Fig.
4) . The portion of azopigment a. 3 not hydrolyzed and the azopigment of bilirubin appear in the organic phase, while the conj ugating lIDieties appear in the water phase. In the initial experiment the azopigment a. was fotmd to be both acid and alkali labile (Table I). 1M NH 3 ). This latter observation was expected since monosaccharides are extremely unstable in alkaline media. However exposure of the azopigment a. 3 to NH3 vapours overnight cleaved the compound to its carboxyllc acid amide and glucose.
Pharmacol Rev 19: 317-366, 1967. 22. MARVER HS, SCHMID R: Biotransformation in the liver: implications for hunan disease. Gastroenterology 55: 282-289, 1968. pigment from herroglobin. The formation of bile Am J Physiol 76: 306-315, 1926. Microsomal heme oxygenase: characterization of the enzyme. J Biol Chern 244: 63886394, 1969. Enzymatic degradation of heme. Oxygenative cleavage requiring cytochrome P-450. Biochemistry 11: 1716-1720, 1972. NADPH-dependent biliverdin reductase: partial purification and characterization.
Jaundice by Antony F. McDonagh (auth.), Carl A. Goresky M.D., Murray M. Fisher M.D. (eds.)
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